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Cereblon-mediated TRPC1 degradation regulates Ca2+ influx in the heart
This study investigated how cereblon (CRBN), a substrate receptor of the E3 ubiquitin ligase complex that selectively degrades ion channels, targets cardiac transient receptor potential channel 1 (TRPC1). Physiologically, CRBN ablation upregulated TRPC1 expression. Mechanistically, the CRBN Lon doma
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