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Modeling the Deleterious Effects of an Endogenously Generated Apolipoprotein-E4 Fragment in Transgenic Zebrafish
Harboring the apolipoprotein E4 allele (APOE4) is the strongest genetic risk factor for late-onset Alzheimer’s disease, yet the mechanisms by which the expressed protein contributes to neurodegeneration remain unknown. Previous studies suggest that proteolytic cleavage of ApoE4 generates amino-termi
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