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UFMylation Suppresses Type I IFN Signaling During M. tuberculosis Infection of Human Macrophages
Type I Interferons (Type I IFNs) are critical for host defense against a wide range of viral infections but can enhance the pathogenicity of several bacterial pathogens, including Mycobacterium tuberculosis (Mtb)(1–4). Given the significance of Type I IFN signaling in determining Mtb infection outco
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