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Intestinal lipid metabolism controls molecular and behavioral immunity through NHR-68 and gut–brain signaling in <i>Caenorhabditis elegans</i>
Animals must allocate limited energetic resources across competing defense programs in response to infection. Here, we show that the conserved nuclear hormone receptor NHR-68 integrates fatty acid metabolism with the neural control of molecular and behavioral immunity in Caenorhabditis elegans. Act
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