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Sph-mediated mtDNA release and autophagy-dependent ferroptosis contribute to <i>Staphylococcus aureus</i>-induced mastitis
Staphylococcus aureus (S. aureus), a major pathogen of mastitis, employs a diverse arsenal of virulence factors to invade and disrupt mammary tissues, thereby eliciting a complex and dysregulated inflammatory response. Sphingomyelin phosphodiesterase (Sph) has emerged as a key virulence determinant
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