NCBI PubMed Central
open access
Recurrent DNA break clusters drive replication-stress-induced copy number variants and genome diversification.
Conceptio Open Archive
Analyze & explore related research in Conceptio
Connect this record directly into your research workflow or AI agent. Explore citations across open repositories, query full text, and export structured proof bundles.
This document is indexed with metadata only: full text is not available in the archive for this record. Open the official source ↗
Related documents
- Targeting PRMTs Creates Vulnerability of DNA Double‐Stand Break Repair Pathways, and Potentiates Chemotherapy Efficacy in TNBC#442577
- Cross-species incompatibilities offer new insights into the functional consequences of satellite DNA evolution.#1107574
- Potential Rad54 separation of function mutation highlights unique roles during homologous recombination#142988
- Potential Rad54 separation of function mutation highlights unique roles during homologous recombination#136459
- BRCA1-RAD51 homologous DNA repair pathway promotes HIV-1 reverse transcription#442561
Record · ID 122885
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Bring this data into your agent: Connect via MCP or API · 20 free credits to start · no card required
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive