HAL (France)
open access
Neuronal A2A receptor exacerbates synapse loss and memory deficits in APP/PS1 mice
Abstract Early pathological upregulation of adenosine A2A receptors (A2ARs), one of the caffeine targets, by neurons is thought to be involved in the development of synaptic and memory deficits in Alzheimer’s disease (AD) but mechanisms remain ill-defined. To tackle this question, we promoted a neur
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