bioRxiv / medRxiv
open access
CC BY 4.0
A Transcritical Bifurcation at Infinity Governs Plaque Stability in a Lipid-Structured Macrophage Model of Atherosclerosis
Atherosclerotic plaques are fatty deposits in arterial walls and a major cause of heart attacks and strokes. Macrophage proliferation triggers plaque growth and instability, but the specific conditions that convert stable plaques into unstable ones remain unclear. To provide insight into the conditi
Conceptio Open Archive
Analyze & explore related research in Conceptio
Connect this record directly into your research workflow or AI agent. Explore citations across open repositories, query full text, and export structured proof bundles.
This document is indexed with metadata only: full text is not available in the archive for this record. Open the official source ↗
Related documents
- Early Therapeutic Effects of Statins and Fibrates on Unstable Atherosclerotic Plaques#992701
- Influence of Lipid Lowering on Impaired Coronary Flow#1195480
- Impact of red blood cell rigidity on in vivo flow dynamics and lingering in bifurcations / Yazdan Rashidi, Felix Maurer, Selina Wrublewsky, Khadija Larhrissi, Thomas John, Frances B. Gidley, Ashley M. Toye, Lars Kaestner, Christian Wagner, Matthias W. Laschke, Alexis Darras#1203156
- Lp-PLA2 and Coronary Atherosclerosis in Humans#827605
- Microglia and CD206+ border-associated mouse macrophages maintain their embryonic origin during Alzheimer’s disease#1203916
Record · ID 1242637
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Bring this data into your agent: Connect via MCP or API · 20 free credits to start · no card required
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive