HAL (France)
open access
Overexpression of Claspin and Timeless protects cancer cells from replication stress in a checkpoint-independent manner
Abstract Oncogene-induced replication stress (RS) promotes cancer development but also impedes tumor growth by activating anti-cancer barriers. To determine how cancer cells adapt to RS, we have monitored the expression of different components of the ATR-CHK1 pathway in primary tumor samples. We sho
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