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The Trp53 delta proline (Trp53ΔP) mouse exhibits increased genome instability and susceptibility to radiation‐induced, but not spontaneous, tumor development
The tumor suppressor TP53 can initiate a plethora of anti-proliferative effects to maintain genomic integrity under conditions of genotoxic stress. The N-terminal proline-rich domain (PRD) of TP53 is important in the regulation of TP53 activity and stability. A common polymorphism at codon 72 in thi
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