eScholarship
metadata only
Enhanced axonal transport: A novel form of “plasticity” after primate and rodent spinal cord injury
Deficient axonal transport after injury is believed to contribute to the failure of CNS regeneration. To better elucidate neural mechanisms associated with CNS responses to injury, we transected the dominant voluntary motor system, the corticospinal tract (CST), in the dorsolateral T10 spinal cord o
Conceptio Open Archive
Analyze & explore related research in Conceptio
Connect this record directly into your research workflow or AI agent. Explore citations across open repositories, query full text, and export structured proof bundles.
This document is indexed with metadata only: full text is not available in the archive for this record. Open the official source ↗
Related documents
- VNS-induced Corticospinal Plasticity#857859
- Emerging strategies for spinal cord injury repair: stem cells, extracellular vesicles, biomaterials, and neuromodulation.#415530
- Cellular Characterization and Neural Electrophysiology in Cerebral Organoid Models of Neurological Disorders.#1299715
- Tissue engineering for traumatic spinal cord injury: Research advances and clinical translation.#402832
- Multiple motor proteins regulate TDP-43 anterograde axonal transport#1312949
Record · ID 1311428
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Bring this data into your agent: Connect via MCP or API · 20 free credits to start · no card required
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive