HAL (France)
open access
Neural serotonergic circuits for controlling long-term voluntary alcohol consumption in mice
Abstract Alcohol-use-disorders are chronic relapsing illnesses, often co-morbid with anxiety. We have previously shown using the “drinking-in-the-dark” model in mice that the stimulation of the serotonin receptor 1A (5-HT 1A ) reduces ethanol binge-drinking behaviour and withdrawal-induced anxiety.
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