HAL (France)
open access
VDAC2 stabilizes a membrane-inserted, primed intermediate of BAX activation
BAX, a major effector of mitochondrial apoptosis, is activated through a series of conformational transitions that lead to mitochondrial outer membrane permeabilization. Genetic studies have established VDAC2 as an essential regulator of BAX-mediated apoptosis, yet the molecular basis of this regula
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