HAL (France)
open access
ZMAT3 alleviates cell death in Fanconi anemia via adaptation of sphingolipid metabolism
Fanconi anemia (FA) is characterized by defective DNA repair and chronic p53 activation, predisposing to acute myeloid leukemia through persistent genomic instability. The molecular adaptations enabling cell survival under chronic stress remain poorly understood. This study investigates the role of
Conceptio Open Archive
Analyze & explore related research in Conceptio
Connect this record directly into your research workflow or AI agent. Explore citations across open repositories, query full text, and export structured proof bundles.
This document is indexed with metadata only: full text is not available in the archive for this record. Open the official source ↗
Related documents
- FUBL-3/FUBP1 mediates mitochondrial stress-induced chromatin remodeling and longevity.#312512
- A high-throughput genetic screening protocol to measure lipid bilayer stress-induced unfolded protein response in Saccharomyces cerevisiae#1133805
- Succinylation-annotated genes in AMI: multi-omics and single-cell prioritization of ASGR2 and NPL.#358274
- Genome-wide machine learning analysis of anosmia and ageusia with COVID-19#453125
- Pervasive cryptic selection in the human noncoding genome#353171
Record · ID 1312989
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Bring this data into your agent: Connect via MCP or API · 20 free credits to start · no card required
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive