University of Nottingham Repository
metadata only
Investigating The Role of Activator Protein -1 Subunits (FOSL2, c-Jun and JunB) in Regulating The Hypoxic Response in Colorectal Cancer (CRC) Cells and Identifying Its Role as a Possible Therapeutic Target For Hypoxic Regions in CRC Tumours
Background: Colorectal cancer (CRC) is the third most common cancer worldwide. Hypoxia (low oxygen) is frequently (~45%) seen in these tumours where it mediates metastasis, resistance to conventional therapy, immunotherapy, and molecular-targeted therapy, resulting in poor patient survival. While hy
Conceptio Open Archive
Analyze & explore related research in Conceptio
Connect this record directly into your research workflow or AI agent. Explore citations across open repositories, query full text, and export structured proof bundles.
This document is indexed with metadata only: full text is not available in the archive for this record. Open the official source ↗
Related documents
- WEE1 kinase inhibition to overcome acquired resistance to targeted therapies in colorectal cancer#480447
- WEE1 kinase inhibition to overcome acquired resistance to targeted therapies in colorectal cancer.#285055
- High INHBB expression in colorectal cancer is associated with poor prognosis and drives malignant phenotypes in tumor cells.#165618
- Oxeiptosis - potential in cancer treatment?#159874
- Investigating the impact of macrophages and hypoxia on cell plasticity in colorectal cancer#287790
Record · ID 1318120
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Bring this data into your agent: Connect via MCP or API · 20 free credits to start · no card required
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive