eScholarship
metadata only
Individual Cardiac Mitochondria Undergo Rare Transient Permeability Transition Pore Openings
RATIONALE: Mitochondria produce ATP, especially critical for survival of highly aerobic cells, such as cardiac myocytes. Conversely, opening of mitochondrial high-conductance and long-lasting permeability transition pores (mPTP) causes respiratory uncoupling, mitochondrial injury, and cell death. Ho
Conceptio Open Archive
Analyze & explore related research in Conceptio
Connect this record directly into your research workflow or AI agent. Explore citations across open repositories, query full text, and export structured proof bundles.
This document is indexed with metadata only: full text is not available in the archive for this record. Open the official source ↗
Related documents
- P-wave Terminal Force in Patients With Atrial Fibrillation.#967405
- Patterns of mitochondrial ATP predict tissue folding.#125436
- Patterns of mitochondrial ATP predict tissue folding#1263865
- Trial of the Effects of Remote Preconditioning on the Tissue Metabolism During Exercise and Ischemia#804471
- Mitochondria directly interact with the nuclear pore complex.#376111
Record · ID 1328473
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Bring this data into your agent: Connect via MCP or API · 20 free credits to start · no card required
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive