bioRxiv / medRxiv
open access
Human genetics implicates a BACH2-NRF2 axis in fetal hemoglobin activation
Human genetic studies have identified key regulators of fetal hemoglobin (HbF) expression, including BCL11A, resulting in therapeutic advances1-8. Yet the mechanisms by which HbF expression is activated remain incompletely understood9. Here, we conduct a large multi-ancestry genome-wide association
Conceptio Open Archive
Analyze & explore related research in Conceptio
Connect this record directly into your research workflow or AI agent. Explore citations across open repositories, query full text, and export structured proof bundles.
This document is indexed with metadata only: full text is not available in the archive for this record. Open the official source ↗
Related documents
- Prenatal Blood Typing With Next Generation Sequencing (NGS) - an Implementation Study in HDFN (PREFAB)#860406
- Association of genetic variation with systolic and diastolic blood pressure among African Americans: the Candidate Gene Association Resource study#1263459
- Gcn5 - mTORC1 - TFEB signalling axis mediated control of autophagy regulates Drosophila blood cell homeostasis#10057
- VIJB: a companion of the JBROWSE genome browser for the visually impaired people.#375451
- Pan-UK Biobank genome-wide association analyses enhance discovery and resolution of ancestry-enriched effects.#219046
Record · ID 1336126
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Bring this data into your agent: Connect via MCP or API · 20 free credits to start · no card required
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive