HAL (France)
open access
Theobroma cacao improves bone growth by modulating defective ciliogenesis in a mouse model of achondroplasia
Abstract A gain-of-function mutation in the fibroblast growth factor receptor 3 gene ( FGFR3 ) results in achondroplasia (ACH), the most frequent form of dwarfism. Constitutive activation of FGFR3 impairs bone formation and elongation and many signal transduction pathways. Identification of new and
Conceptio Open Archive
Analyze & explore related research in Conceptio
Connect this record directly into your research workflow or AI agent. Explore citations across open repositories, query full text, and export structured proof bundles.
This document is indexed with metadata only: full text is not available in the archive for this record. Open the official source ↗
Related documents
- Immunometabolic pathways in liver fibrosis: current evidence for selected plant-derived metabolites associated with traditional Chinese medicine.#469162
- A switch in collagen expression regulates cessation of distal tip cell migration in C. elegans#1250722
- Multi-ligand simultaneous docking of Carica papaya leaf phytochemicals, Carpaine and Rutin, reveals multi-mechanism inhibition of cancer proteins BCL-2 and WWP1#985891
- Loss of MITF activity leads to emergent cell states from the melanocyte stem cell lineage#172231
- Author Correction: A human progeria-associated BAF-1 mutation modulates gene expression and accelerates aging in C. elegans#1250046
Record · ID 1345778
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Bring this data into your agent: Connect via MCP or API · 20 free credits to start · no card required
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive