Lund University Publications
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Rac1 signaling regulates neutrophil-dependent tissue damage in experimental colitis.
Excessive neutrophil recruitment in the colon is a major feature in acute colitis although the signaling mechanisms behind colonic recruitment of neutrophils remain elusive. Herein, we hypothesized that Rac1 activity might play an important role in neutrophil infiltration in the inflamed colon. Fema
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