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Elevated MARCKS phosphorylation contributes to unresponsiveness of breast cancer to paclitaxel treatment
Accumulating evidence has suggested that myristoylated alanine-rich C-kinase substrate (MARCKS) is critical for regulating multiple pathophysiological processes. However, the molecular mechanism underlying increased phosphorylation of MARCKS at Ser159/163 (phospho-MARCKS) and its functional conseque
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