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The role of protein kinase B/Akt in insulin-induced inactivation of phosphorylase in rat hepatocytes
Aims/hypothesis: An insulin signalling pathway leading from activation of protein kinase B (PKB, also known as Akt) to phosphorylation (inactivation) of glycogen synthase kinase-3 (GSK-3) and activation of glycogen synthase is well characterised. However, in hepatocytes, inactivation of GSK-3 is not
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