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Antagonizing CCR2 with propagermanium leads to altered distribution of macrophage subsets and favorable tissue remodeling after myocardial infarction in mice
Aims: The aim of the present study was to investigate the inhibition of classically activated macrophages in myocardial infarction (MI) under the influence of the chemokine (C-C motif) receptor 2 (CCR2) antagonist propagermanium (PPG). Methods and Results: Mice (C57BL/6; n = 121) were subjected to
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