HAL (France)
open access
Evidence for similar enzymatic but different cellular generation of pE79-modified α-synuclein in PD and AD mouse models
The α-synuclein (aSyn) pathology is a predominant feature of synucleinopathies such as Parkinson's disease (PD). Triggers that may induce aSyn aggregation are not completely understood, but include post-translational modifications. One such modification is aSyn truncation exposing the N-terminal glu
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