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Immune mitochondrial complex I mediates vascular damage in toll-like receptor 7 (TLR7)-induced lupus.
Systemic lupus erythematosus (SLE) is a chronic autoimmune disease characterized by immune dysregulation and high cardiovascular risk, including hypertension and endothelial dysfunction. Metabolic reprogramming of immune cells, particularly CD4 + T cells, contributes to SLE pathogenesis. We investig
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