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<b>Systematic analysis reveals an Mln1-dependent trade-off between metabolic flexibility and thermotolerance in </b><b><i>Cryptococcus neoformans</i></b>
Fungal pathogens entering a mammalian host encounter elevated temperature, nutrient limitation, and immune stress simultaneously, making it difficult to assign specific regulators to specific cues. Here we separate a host-mimicking condition into its component signals to define host-res
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