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<b>Short-term </b><b><i>in vivo</i></b><b> exposure to murine lung neutrophils of differing oxidative capacity does not drive detectable genomic change in </b><b><i>Aspergillus fumigatus</i></b>
Neutrophil NADPH-oxidase (NOX)-derived reactive oxidative stress contributes to pulmonary clearance of Aspergillus fumigatus and is assumed to impose a strong selective pressure on inhaled conidia. It, however, remains unclear if repeated exposure to host neutrophil oxidative kil
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