bioRxiv / medRxiv
open access
CC BY-NC 4.0
Multi-omics profiling reveals MAGEL2-driven defects in human corticogenesis shared across Prader-Willi and Schaaf-Yang syndromes.
Conceptio Open Archive
Analyze & explore related research in Conceptio
Connect this record directly into your research workflow or AI agent. Explore citations across open repositories, query full text, and export structured proof bundles.
This document is indexed with metadata only: full text is not available in the archive for this record. Open the official source ↗
Related documents
- RAI1 safeguards fidelity and tempo of human neurodevelopmental gene expression#359645
- "SHANK3 deficiency alters early progenitor dynamics and reveals shared pathways with neurodegeneration"#224200
- Human-specific gene expansions contribute to brain evolution.#388809
- Pathogenic variants in KMT2C result in a neurodevelopmental disorder distinct from Kleefstra and Kabuki syndromes#824011
- Transcriptional repression by TGIF2 coordinates neurogenic priming and neural stem cell maintenance#823797
Record · ID 252840
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Bring this data into your agent: Connect via MCP or API · 20 free credits to start · no card required
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive