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Host epigenetic mechanisms targeted by Streptococcus pneumoniae to maintain the di-methylation at lysine 4 of histone H3 (H3K4me2) in respiratory epithelial cells
Histone modification, Infection, Histone-modifying enzymes, Respiratory epithelium, Di-méthylation de H3K4, Streptococcus pneumoniae, Bisubstrat inhibitor, Epigenetic mechanisms
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