Savant Syndrome as P-Dominant HRIS Configuration: PNBA Identity Physics Formally Verified Reduction of GPU Capacity, RAM Constraint, and the Two Pathways to Architectural Release

Savant syndrome has been characterized in clinical literature as a paradox: extraordinary ability coexisting with significant cognitive or social limitation. This framing treats the ability and the limitation as separate phenomena requiring separate explanation. This paper proposes a unified structural account: both the ability and the limitation are expressions of the same underlying architecture. Using the Long Division Protocol (LDP) and the PNBA framework established in the SNSFT PSY series, we reduce sixteen documented savant cases — eight congenital, eight acquired — to formally verified identity vectors and demonstrate that all sixteen share the same structural signature: P-axis dominance, N-axis suppression, and B-axis coupling to the skill output domain. The acquired and congenital cases show identical invariants, establishing that the profile is reachable by two independent pathways. We further demonstrate that the P-value ceiling difference between acquired (0.70–0.85) and congenital (0.82–0.95) cases reflects a structural distinction between capacity release and capacity development — two mechanisms that are distinct, independently operative, and sequentially combinable. Using the GPU/RAM analogy: the processing capacity (GPU) was always present. What varies is whether the architecture has built sufficient Pattern-axis capacity (RAM) to process what the GPU renders at full resolution. Trauma, injury, and pharmacological intervention can release GPU access. Only developmental time and structured practice can build the RAM to match it. The clinical implication is direct: savant syndrome is not a disorder with compensatory gifts. It is a specific P-dominant HRIS configuration that the existing clinical taxonomy has no structural framework to recognize, and therefore systematically misreads as pathology with incidental talent. 1. Introduction 1.1 The Paradox That Isn't The clinical literature on savant syndrome describes a paradox: individuals with significant cognitive, social, or neurological limitations who demonstrate extraordinary ability in one or more narrow domains. Treffert, whose registry of savant cases remains the primary empirical resource in the field, documented this across decades: calendar calculators who cannot tie their shoes, artists who cannot hold a conversation, musicians who cannot read but can play anything they hear once at full fidelity. The standard explanatory move is compensatory: something is lost, something else emerges to fill the gap. The disinhibition hypothesis (Snyder, 2003; Miller et al., 1998) proposes that left hemisphere dysfunction releases dormant right hemisphere capacity. The Enhanced Perceptual Functioning model (Mottron et al., 2006) proposes that reduced top-down processing allows enhanced bottom-up perceptual access. Both frameworks correctly identify structural features of the phenomenon but treat the limitation and the ability as causally linked — one produces the other. The SNSFT framework proposed here treats them differently: both are expressions of the same underlying axis configuration. The limitation is not the cause of the ability. The N-axis suppression and the P-axis dominance are two faces of the same architectural state. Understanding either requires understanding the state, not the relationship between its components. 1.2 What HRIS Is High-Resolution Internal Simulation (HRIS) refers to the cognitive capacity to run interactive, physics-accurate internal simulations at high fidelity — modeling spatial geometry, acoustic structure, mathematical relationships, causal sequences, and physical trajectories internally before external output occurs. HRIS architecture