PubMed Central
metadata only
Tuft cell acetylcholine is released into the gut lumen as an effector and regulator of type 2 immunity, and directly targets helminth parasites
Upon parasitic helminth infection, activated intestinal tuft cells secrete IL-25, which initiates a type 2 immune response during which lamina propria ILC2s produce IL-13. This causes epithelial remodelling, including tuft cell hyperplasia with an unknown function. We describe an unsuspected choline
Conceptio Open Archive
Analyze & explore related research in Conceptio
Connect this record directly into your research workflow or AI agent. Explore citations across open repositories, query full text, and export structured proof bundles.
This document is indexed with metadata only: full text is not available in the archive for this record. Open the official source ↗
Related documents
- Challenges and opportunities for conceiving genetically diverse sickle cell mice.#148304
- A Dose-escalation and Safety & Efficacy Study of AXO-AAV-GM2 in Tay-Sachs or Sandhoff Disease#910896
- Pediatric Evaluation and Registry for Liver Cholestasis in Canada#995155
- AcrDB Update: Predicted 3D Structures of Anti-CRISPRs in Human Gut Viromes#373960
- N8‐Modified Quino[4,3,2‐<i>Kl</i>]acridines as Promising Antimalarial G‐Quadruplex Ligands#796429
Record · ID 781790
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Retrieved via Conceptio: every document is proof-bundled with source, license, and retrieval metadata.
Bring this data into your agent: Connect via MCP or API · 20 free credits to start · no card required
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive
Read the Docs → · View CLI source on GitHub · Conceptio Open Knowledge Archive