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Disruption of sphingolipid metabolism promotes tau seeding through endolysosomal membrane rigidification and rupture
Endolysosomal dysfunction is a hallmark of Alzheimer’s disease (AD) and related tauopathies, yet underlying mechanisms remain poorly understood. This study investigates the role of sphingolipid metabolism in maintaining endolysosomal membrane integrity and its impact on tau aggregation and toxicity
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