Repositorio de Universidad Autónoma de Chile.
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Gap-junctional channel and hemichannel activity of two recently identified connexin 26 mutants associated with deafness
Gap-junction channels (GJCs) are formed by head-to-head association of two hemichannels (HCs, connexin hexamers). HCs and GJCs are permeable to ions and hydrophilic molecules of up to Mr ~1 kDa. Hearing impairment of genetic origin is common, and mutations of connexin 26 (Cx26) are its major cause.
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