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Influence of PARP1 on CRISPR\/Cas9 induced double strand break repair in proliferating cells
Genome editing with CRISPR/Cas9 depends on the induction of site-specific DNA double-strand breaks (DSBs), which are repaired by distinct pathways. Non-homologous end joining (NHEJ) frequently introduces insertions or deletions (indels), resulting in mutagenic repair, whereas microhomology-mediated
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