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RAD51 stabilizes neutrophil extracellular traps to compartmentalize inflammation
Neutrophil extracellular traps (NETs) feature a branched chromatin architecture whose origin and function remain unknown. We found that NET branching is mediated by RAD51, a protein generating DNA junctions during DNA recombination repair. Pharmacological inhibition, RAD51 knock-down or GEN1 and Ruv
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